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dc.contributor.authorP. Tanphaichitren_US
dc.contributor.authorD. Tanphaichitren_US
dc.contributor.authorJ. Sureeratananen_US
dc.contributor.authorS. Chatasinghen_US
dc.contributor.otherMahidol Universityen_US
dc.date.accessioned2018-04-30T08:37:26Z-
dc.date.available2018-04-30T08:37:26Z-
dc.date.issued1980-01-01en_US
dc.identifier.citationThe Journal of Pediatrics. Vol.96, No.3 PART 1 (1980), 490-493en_US
dc.identifier.issn00223476en_US
dc.identifier.other2-s2.0-0018830936en_US
dc.identifier.urihttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=0018830936&origin=inwarden_US
dc.identifier.urihttp://repository.li.mahidol.ac.th/dspace/handle/123456789/11160-
dc.description.abstractConsiderable evidence suggests a role of abnormal T-cell lymphocyte functions in the pathogenesis of minimal lesion nephrotic syndrome. The mean ± SD T-cell lymphocytes as determined by %E-rosettes among 10 children after 24 to 84 months of complete remission was 66.7±4.5; this is statistically different from that of seven children with minimal lesion nephrotic syndrome during relapse, 33.5±9.5 (P < 0.01). After levamisole therapy at 1.5 to 3.9 mg/kg/dose twice weekly for one to six months, the mean ±SD %E-rosettes in the latter group was 69.3±3.9, which is not statistically different from that in the group with complete remission after conventional treatment with steroids. Those treated with levamisole also had a complete remission without any side effects. © 1980 The C. V. Mosby Company.en_US
dc.rightsMahidol Universityen_US
dc.source.urihttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=0018830936&origin=inwarden_US
dc.subjectMedicineen_US
dc.titleTreatment of nephrotic syndrome with levamisoleen_US
dc.typeArticleen_US
dc.rights.holderSCOPUSen_US
dc.identifier.doi10.1016/S0022-3476(80)80707-4en_US
Appears in Collections:Scopus 1969-1990

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