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dc.contributor.authorSuthat Fucharoenen_US
dc.contributor.authorP. Winichagoonen_US
dc.contributor.otherMahidol Universityen_US
dc.date.accessioned2018-06-11T04:40:01Z-
dc.date.available2018-06-11T04:40:01Z-
dc.date.issued2012-01-01en_US
dc.identifier.citationInternational Journal of Laboratory Hematology. Vol.34, No.6 (2012), 559-565en_US
dc.identifier.issn1751553Xen_US
dc.identifier.issn17515521en_US
dc.identifier.other2-s2.0-84873314430en_US
dc.identifier.urihttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=84873314430&origin=inwarden_US
dc.identifier.urihttp://repository.li.mahidol.ac.th/dspace/handle/123456789/13831-
dc.description.abstractThalassemia and abnormal hemoglobin are the most common genetic disorders and are considered health problems in many developing countries. In the last few years, there has been much progress in laboratory diagnosis, treatment and control of thalassemia. The variation in the clinical severity in both a- and b-thalassemia reflects a genotype-phenotype interaction. This is important for future therapeutic intervention and should be well characterized in each population. The quality of life of the patients is much improved with regular blood transfusion and novel iron chelators. The cure for thalassemia is possible by stem cell transplantation and future gene therapy. It is expected that under multinational collaboration the prevention of thalassemia will happen worldwide. © 2012 Blackwell Publishing Ltd.en_US
dc.rightsMahidol Universityen_US
dc.source.urihttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=84873314430&origin=inwarden_US
dc.subjectBiochemistry, Genetics and Molecular Biologyen_US
dc.subjectMedicineen_US
dc.titleNew updating into hemoglobinopathiesen_US
dc.typeReviewen_US
dc.rights.holderSCOPUSen_US
dc.identifier.doi10.1111/j.1751-553X.2012.01446.xen_US
Appears in Collections:Scopus 2011-2015

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