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dc.contributor.authorS. N. Wickramasingheen_US
dc.contributor.authorSornchai Looareesuwanen_US
dc.contributor.authorBussarin Nagachintaen_US
dc.contributor.authorN. J. Whiteen_US
dc.contributor.otherSt Mary's Hospital Londonen_US
dc.contributor.otherMahidol Universityen_US
dc.contributor.otherPaholpolpayuhasena Hospitalen_US
dc.contributor.otherUniversity of Oxforden_US
dc.identifier.citationBritish Journal of Haematology. Vol.72, No.1 (1989), 91-99en_US
dc.description.abstractSummary. Nine Thai adults with P. vivax malaria were investigated. Light and electron microscope studies of marrow aspirates revealed morphological evidence of dyserythro‐poiesis in six of them, Dyserythropoiesis was most marked in the four most anaemic patients. In these four patients the electron microscope also revealed the presence of erythro‐blasts at various stages of degradation within the cytoplasm of macrophages. Neither the dyserythropoiesis nor the ineffective erythropoiesis could be attributed to a deficiency of vitamin B 12 , folate or iron. The abnormalities of erythropoiesis seemed to result from the P. vivax infection itself. Other bone marrow reactions seen in this infection included macrophage hyperplasia, plasmacytosis and increased eosinophil granulocytopoiesis. Unlike in severe P. falciparum malaria, the microvasculature of the marrow was not obstructed by parasitized red cells. Copyright © 1989, Wiley Blackwell. All rights reserveden_US
dc.rightsMahidol Universityen_US
dc.titleDyserythropoiesis and ineffective erythropoiesis in Plasmodium vivax malariaen_US
Appears in Collections:Scopus 1969-1990

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