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dc.contributor.authorY. Metugriachuken_US
dc.contributor.authorF. Marottaen_US
dc.contributor.authorK. Pavasuthipaisiten_US
dc.contributor.authorO. Kuroien_US
dc.contributor.authorJ. Tsuchiyaen_US
dc.contributor.authorA. Lorenzettien_US
dc.contributor.authorE. Fesceen_US
dc.contributor.authorE. Minellien_US
dc.contributor.otherTMC Hospitalen_US
dc.contributor.otherBiokenkyusho Research Laboratoryen_US
dc.contributor.otherS. Giuseppe Hospitalen_US
dc.contributor.otherUniversita degli Studi di Milanoen_US
dc.contributor.otherMahidol Universityen_US
dc.identifier.citationRejuvenation Research. Vol.9, No.2 (2006), 342-345en_US
dc.description.abstractMotility recording of small and large intestine was performed in old Wistar rats divided into three groups: (a) standard diet, (b) standard diet plus a symbiotic preparation, and (c) standard diet plus a heat-inactivated symbiotic preparation. SCM-III. significantly increased the myoelectric activity of small intestine and colon (p < 0.01 versus [a] and [c]) paralleling "young" values of 4-month-old rats and increased the spike burst frequency in the proximal-distal colon (p < 0.05). SCM-III significantly increased the frequency and duration of spike bursts in the jejunum, transverse-distal colon, and defecation frequency, while decreasing the intervals of migrating motor complex in the colon (p < 0.01) to "young" values with an increased mRNA expression of VIP (p < 0.05). Gut flora manipulation aimed to modulate myoelectric activity can tentatively help reversing age-related motility decay. © Mary Ann Liebert, Inc.en_US
dc.rightsMahidol Universityen_US
dc.subjectBiochemistry, Genetics and Molecular Biologyen_US
dc.titleThe aging gut motility decay: May symbiotics be acting as "implantable" biologic pace-makers?en_US
dc.typeConference Paperen_US
Appears in Collections:Scopus 2006-2010

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