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dc.contributor.authorArada Rojana-Udomsarten_US
dc.contributor.authorChalermchai Mitrpanten_US
dc.contributor.authorIan Jamesen_US
dc.contributor.authorCampbell Witten_US
dc.contributor.authorMerrilee Needhamen_US
dc.contributor.authorTimothy Dayen_US
dc.contributor.authorLynette Kiersen_US
dc.contributor.authorAlastair Corbetten_US
dc.contributor.authorPatricia Martinezen_US
dc.contributor.authorSteve D. Wiltonen_US
dc.contributor.authorFrank L. Mastagliaen_US
dc.contributor.otherUniversity of Western Australiaen_US
dc.contributor.otherYala Hospitalen_US
dc.contributor.otherMahidol Universityen_US
dc.contributor.otherRoyal Perth Hospitalen_US
dc.contributor.otherRoyal North Shore Hospitalen_US
dc.contributor.otherUniversity of Sydney Faculty of Medicineen_US
dc.contributor.otherRoyal Melbourne Hospitalen_US
dc.contributor.otherUniversity of Melbourneen_US
dc.contributor.otherConcord Repatriation General Hospitalen_US
dc.identifier.citationJournal of Neuroimmunology. Vol.254, No.1-2 (2013), 174-177en_US
dc.description.abstractWe compared the carriage frequencies of HLA-DRB3 and its major alleles and of HLA-DRB4 and HLA-DRB5 in an Australian sIBM cohort and a population control group who had previously been genotyped for the HLA-DRB1*03:01 risk allele. There was a strong disease association with the carriage of the DRB3*01:01 allele which was accounted for by its linkage disequilibrium with DRB1*03:01. The carriage of HLA-DRB4 was found to be strongly protective and abrogated the risk effect of HLA-DRB1*03:01. The findings indicate that haplotypic combinations of alleles at the HLA-DRB1 and secondary HLA-DRB loci have important risk modifying effects in sIBM. © 2012 Elsevier B.V.en_US
dc.rightsMahidol Universityen_US
dc.subjectImmunology and Microbiologyen_US
dc.titleAnalysis of HLA-DRB3 alleles and supertypical genotypes in the MHC Class II region in sporadic inclusion body myositisen_US
Appears in Collections:Scopus 2011-2015

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