Mallika ImwongSasithon PukrittayakameeWirichada PongtavornpinyoSupatchara NakeesathitShalini NairPaul NewtonFrancois NostenTimothy J.C. AndersonArjen DondorpNicholas P.J. DayNicholas J. WhiteMahidol UniversityRoyal InstituteTexas Biomedical Research InstituteWellcome Trust-Mahosot Hospital-Oxford Tropical Medicine Research CollaborationShoklo Malaria Research UnitChurchill Hospital2018-07-122018-07-122008-07-01Antimicrobial Agents and Chemotherapy. Vol.52, No.7 (2008), 2657-2659006648042-s2.0-46249096629https://repository.li.mahidol.ac.th/handle/20.500.14594/19630Plasmodium vivax mdr1 gene amplification, quantified by real-time PCR, was significantly more common on the western Thailand border (6 of 66 samples), where mefloquine pressure has been intense, than elsewhere in southeast Asia (3 of 149; P = 0.02). Five coding mutations in pvmdr1, independent of gene amplification, were also found. Copyright © 2008, American Society for Microbiology. All Rights Reserved.Mahidol UniversityMedicinePharmacology, Toxicology and PharmaceuticsArticleSCOPUS10.1128/AAC.01459-07