S. FucharoenP. WinichagoonS. ChaicharoenP. WasiMahidol University2018-06-142018-06-141987-01-01European Journal of Haematology. Vol.39, No.2 (1987), 154-16016000609090244412-s2.0-0023388555https://repository.li.mahidol.ac.th/handle/20.500.14594/15444DNA from members of 2 Thai families with conditions considered to be δβ‐thalassaemia were studied by using restriction endonuclease DNA mapping. The propositus in family A is a double heterozygote for β‐thalassaemia and δβ‐thalassaemia. DNA analysis reveals a deletion of the β‐globin gene cluster starting at the area between the Sac I and Eco RI sites near the 3′ end of the G γ‐gene and extending through the A γ‐, δ‐ and β‐genes to an unknown extent downstream. In family B, the propositus is δβ‐thalassaemia/Hb E. Deletion of the β‐globin gene cluster begins in the large intervening sequence of the A γ‐gene and removes both δ‐ and β‐genes downstream. © Munksgaard 1987Mahidol UniversityMedicineDifferent molecular defects of <sup>G</sup>γ (<sup>A</sup>γδβ)°‐thalassaemia in ThailandArticleSCOPUS10.1111/j.1600-0609.1987.tb00746.x