Publication: Urticarial vasculitis: Etiologies and clinical course
Issued Date
2009-06-01
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ISSN
0125877X
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2-s2.0-70350724547
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Mahidol University
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SCOPUS
Bibliographic Citation
Asian Pacific Journal of Allergy and Immunology. Vol.27, No.2-3 (2009), 95-102
Suggested Citation
Kanokvalai Kulthanan, Meethawee Cheepsomsong, Sukhum Jiamton Urticarial vasculitis: Etiologies and clinical course. Asian Pacific Journal of Allergy and Immunology. Vol.27, No.2-3 (2009), 95-102. Retrieved from: https://repository.li.mahidol.ac.th/handle/123456789/27705
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Title
Urticarial vasculitis: Etiologies and clinical course
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Abstract
Out of 64 patients diagnosed with urticarial vasculitis (UV), 49 (76.6%) presented with their first attack of UV. The others experienced recurrent attacks with a mean number of 3.3 past recurrences. Fifteen patients had angioedema (23.4%) and 16 (25%) suffered systemic involvement. The most common abnormal laboratory finding was an increased erythrocyte sedimentation rate. Six of 62 patients (9.7%) had decreased C3 levels. A cause could be identified in 19 patients (29.7%). The most common identified cause was infection; other causes included drugs, malignancy and systemic lupus erythematosus (SLE). The prevalence of immunoreactant deposits in the skin lesions measured by DIF was 54.7% (35 of 64 patients). The median disease duration of each episode was 85 days. The probability that patients were free of symptoms within one year was 70%. Patients with an idiopathic cause had a statistically significant longer course duration of each episode than the group with upper respiratory tract infection. Compared to reports from Western countries, our patients seemed to have less severe symp-toms and a lower percentage of hypocomplementemic UV and SLE.