Publication:
Monosodium glutamate-induced oxidative kidney damage and possible mechanisms: a mini-review

dc.contributor.authorAmod Sharmaen_US
dc.contributor.otherMahidol University. Siriraj Hospital. Department of Physiologyen_US
dc.date.accessioned2017-08-04T07:00:21Z
dc.date.available2017-08-04T07:00:21Z
dc.date.created2017-08-04
dc.date.issued2015
dc.description.abstractAnimal studies suggest that chronic monosodium glutamate (MSG) intake induces kidney damage by oxidative stress. However, the underlying mechanisms are still unclear, despite the growing evidence and consensus that α-ketoglutarate dehydrogenase, glutamate receptors and cystine-glutamate antiporter play an important role in up-regulation of oxidative stress in MSG-induced renal toxicity. This review summaries evidence from studies into MSG-induced renal oxidative damage, possible mechanisms and their importance from a toxicological viewpoint.en_US
dc.identifier.citationJournal of Biomedical Science. Vol. 22, (2015), 93en_US
dc.identifier.doi10.1186/s12929-015-0192-5
dc.identifier.urihttps://repository.li.mahidol.ac.th/handle/123456789/2667
dc.language.isoengen_US
dc.rightsMahidol Universityen_US
dc.rights.holderBioMed Centralen_US
dc.subjectOpen Access articleen_US
dc.subjectMonosodium glutamateen_US
dc.subjectKidneyen_US
dc.subjectOxidative stressen_US
dc.subjectGlutamate receptorsen_US
dc.subjectα-Ketoglutarate dehydrogenaseen_US
dc.titleMonosodium glutamate-induced oxidative kidney damage and possible mechanisms: a mini-reviewen_US
dc.typeReview Articleen_US
dspace.entity.typePublication

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