Methacrylic acid/Tween 80 engineered amorphous calcium phosphate as an effective bioactive reinforcing nanofillers in dental adhesive
1
Issued Date
2025-01-01
Resource Type
ISSN
02874547
eISSN
18811361
Scopus ID
2-s2.0-105002282203
Journal Title
Dental Materials Journal
Volume
44
Issue
2
Start Page
190
End Page
200
Rights Holder(s)
SCOPUS
Bibliographic Citation
Dental Materials Journal Vol.44 No.2 (2025) , 190-200
Suggested Citation
Maimansomsuk S., Teangrom P., Teanchai C., Sinthuvanich C., Ruengket P., Attarataya J., Akkarachaneeyakorn K. Methacrylic acid/Tween 80 engineered amorphous calcium phosphate as an effective bioactive reinforcing nanofillers in dental adhesive. Dental Materials Journal Vol.44 No.2 (2025) , 190-200. 200. doi:10.4012/dmj.2024-285 Retrieved from: https://repository.li.mahidol.ac.th/handle/123456789/109536
Title
Methacrylic acid/Tween 80 engineered amorphous calcium phosphate as an effective bioactive reinforcing nanofillers in dental adhesive
Corresponding Author(s)
Other Contributor(s)
Abstract
Bioactive fillers in dental adhesives are designed to release beneficial ions, such as calcium and phosphate, to help in remineralization process, and preventing secondary caries. Their incorporation also aims to improve the longevity of dental restorations. Herein, CaP nanoparticles were synthesized through a water-in-oil emulsion method using Tween80 as a surfactant and methacrylic acid (MAA). MAA officiated as a co-surfactant and reduced the size of CaP nanoparticle, including improved compatibility with the components of dental adhesive. The incorporation of 2% T80/CaP/MAA nanofillers into dental adhesive demonstrated increased efficiency, as evidenced by a higher degree of conversion (%DC) and greater micro-tensile bond strength (μTBS). More importantly, T80/CaP/ MAA nanofillers were able to induce remineralization process in the dental structure, which was confirmed by EDX mapping and IR imaging. Finally, a cytotoxicity assessment of the dental adhesive containing T80/CaP/MAA showed no significant adverse effects on human fibroblast cells.