Publication:
Immunopathogenesis of Dengue Virus-Induced Redundant Cell Death: Apoptosis and Pyroptosis

dc.contributor.authorSan Suwanmaneeen_US
dc.contributor.authorNatthanej Luplertlopen_US
dc.contributor.otherMahidol Universityen_US
dc.date.accessioned2018-12-21T06:57:12Z
dc.date.accessioned2019-03-14T08:03:03Z
dc.date.available2018-12-21T06:57:12Z
dc.date.available2019-03-14T08:03:03Z
dc.date.issued2017-01-01en_US
dc.description.abstractCopyright © 2017, Mary Ann Liebert, Inc. 2017. Dengue virus infection is a self-limited condition, which is of particular importance in tropical and subtropical regions and for which no specific treatment or effective vaccine is available. There are several hypotheses explaining dengue pathogenesis. These usually refer to host immune responses, including antibody-dependent enhancement, cytokine expression, and dengue virus particles including NS1 protein, which lead to cell death by both apoptosis and pyroptosis. A clear understanding of the pathogenesis should facilitate the development of vaccines and therapies. This review focuses on the immunopathogenesis in relation to clinical manifestations and patterns of cell death, focusing on the pathogenesis of severe dengue.en_US
dc.identifier.citationViral Immunology. Vol.30, No.1 (2017), 13-19en_US
dc.identifier.doi10.1089/vim.2016.0092en_US
dc.identifier.issn15578976en_US
dc.identifier.issn08828245en_US
dc.identifier.other2-s2.0-85009909977en_US
dc.identifier.urihttps://repository.li.mahidol.ac.th/handle/20.500.14594/42017
dc.rightsMahidol Universityen_US
dc.rights.holderSCOPUSen_US
dc.source.urihttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85009909977&origin=inwarden_US
dc.subjectBiochemistry, Genetics and Molecular Biologyen_US
dc.titleImmunopathogenesis of Dengue Virus-Induced Redundant Cell Death: Apoptosis and Pyroptosisen_US
dc.typeArticleen_US
dspace.entity.typePublication
mu.datasource.scopushttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85009909977&origin=inwarden_US

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