Publication: Platelet function tests in thalassemic children.
| dc.contributor.author | P. Isarangkura | en_US |
| dc.contributor.author | P. Pintadit | en_US |
| dc.contributor.author | P. Hathirat | en_US |
| dc.contributor.author | W. Sasanakul | en_US |
| dc.contributor.other | Mahidol University | en_US |
| dc.date.accessioned | 2018-06-14T09:00:45Z | |
| dc.date.available | 2018-06-14T09:00:45Z | |
| dc.date.issued | 1987-12-01 | en_US |
| dc.description.abstract | Platelet changes in thalassemia included prolongation of bleeding time (30%), thrombocytosis (29%), decreased platelet retention (79%), and normal PF3 release. Platelet aggregation was found to be hyperaggregation in 50%, normal in 22%, and hypoaggregation in 28% of the studied patients. Platelet changes in thalassemia can be hyper-, normal, or hypofunction. The changes are likely to be related to many factors, particularly to the progress of the disease. Platelet hypoaggregation may lead to a bleeding problem. Platelet hyperaggregation and thrombocytosis were prominent in the splenectomized patients and/or the severely anemic group; antiplatelet drugs may be indicated in these patients. | en_US |
| dc.identifier.citation | Birth defects original article series. Vol.23, No.5 A (1987), 395-401 | en_US |
| dc.identifier.issn | 05476844 | en_US |
| dc.identifier.other | 2-s2.0-0023474259 | en_US |
| dc.identifier.uri | https://repository.li.mahidol.ac.th/handle/20.500.14594/15301 | |
| dc.rights | Mahidol University | en_US |
| dc.rights.holder | SCOPUS | en_US |
| dc.source.uri | https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=0023474259&origin=inward | en_US |
| dc.subject | Biochemistry, Genetics and Molecular Biology | en_US |
| dc.subject | Medicine | en_US |
| dc.title | Platelet function tests in thalassemic children. | en_US |
| dc.type | Article | en_US |
| dspace.entity.type | Publication | |
| mu.datasource.scopus | https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=0023474259&origin=inward | en_US |